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Artigo de periodico
Codon 129 polymorphism of prion protein gene in is not a risk factor for Alzheimer's disease (2013)
Smid, Jerusa; Landemberger, Michele Christine; Bahia, Valeria Santoro; Martins, Vilma Regina; Nitrini, Ricardo
Source: Arquivos de Neuro-Psiquiatria. Unidade: FM
Subjects: DOENÇA DE ALZHEIMER (ETIOLOGIA;DIAGNÓSTICO;GENÉTICA), POLIMORFISMO, IDOSOS, GENÓTIPOS, BRASILEIROS, ALELOPATIA
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ABNT
SMID, Jerusa et al. Codon 129 polymorphism of prion protein gene in is not a risk factor for Alzheimer's disease. Arquivos de Neuro-Psiquiatria, v. 71, n. 7, p. 423-427, 2013Tradução . . Disponível em: https://doi.org/10.1590/0004-282X20130055. Acesso em: 29 maio 2024.
APA
Smid, J., Landemberger, M. C., Bahia, V. S., Martins, V. R., & Nitrini, R. (2013). Codon 129 polymorphism of prion protein gene in is not a risk factor for Alzheimer's disease. Arquivos de Neuro-Psiquiatria, 71( 7), 423-427. doi:10.1590/0004-282X20130055
NLM
Smid J, Landemberger MC, Bahia VS, Martins VR, Nitrini R. Codon 129 polymorphism of prion protein gene in is not a risk factor for Alzheimer's disease [Internet]. Arquivos de Neuro-Psiquiatria. 2013 ; 71( 7): 423-427.[citado 2024 maio 29 ] Available from: https://doi.org/10.1590/0004-282X20130055
Vancouver
Smid J, Landemberger MC, Bahia VS, Martins VR, Nitrini R. Codon 129 polymorphism of prion protein gene in is not a risk factor for Alzheimer's disease [Internet]. Arquivos de Neuro-Psiquiatria. 2013 ; 71( 7): 423-427.[citado 2024 maio 29 ] Available from: https://doi.org/10.1590/0004-282X20130055
Artigo de periodico
Cellular prion protein modulates defensive attention and innate fear-induced behaviour evoked in transgenic mice submitted to an agonistic encounter with the tropical coral snake Oxyrhopus guibei (2008)
Lobão-Soares, Bruno; Walz, Roger; Prediger, Rui Daniel Schröder; Freitas, Renato Leonardo; Calvo, Fabrício; Muxfeldt, Marino; Leite, João Pereira; Landemberger, Michele Christine; Coimbra, Norberto Cysne
Source: Behavioural Brain Research. Unidade: FMRP
Subjects: GLICOPROTEÍNAS, NEUROBIOLOGIA
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ABNT
LOBÃO-SOARES, Bruno et al. Cellular prion protein modulates defensive attention and innate fear-induced behaviour evoked in transgenic mice submitted to an agonistic encounter with the tropical coral snake Oxyrhopus guibei. Behavioural Brain Research, v. 194, n. 2, p. 129-137, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2008.06.006. Acesso em: 29 maio 2024.
APA
Lobão-Soares, B., Walz, R., Prediger, R. D. S., Freitas, R. L., Calvo, F., Muxfeldt, M., et al. (2008). Cellular prion protein modulates defensive attention and innate fear-induced behaviour evoked in transgenic mice submitted to an agonistic encounter with the tropical coral snake Oxyrhopus guibei. Behavioural Brain Research, 194( 2), 129-137. doi:10.1016/j.bbr.2008.06.006
NLM
Lobão-Soares B, Walz R, Prediger RDS, Freitas RL, Calvo F, Muxfeldt M, Leite JP, Landemberger MC, Coimbra NC. Cellular prion protein modulates defensive attention and innate fear-induced behaviour evoked in transgenic mice submitted to an agonistic encounter with the tropical coral snake Oxyrhopus guibei [Internet]. Behavioural Brain Research. 2008 ; 194( 2): 129-137.[citado 2024 maio 29 ] Available from: https://doi.org/10.1016/j.bbr.2008.06.006
Vancouver
Lobão-Soares B, Walz R, Prediger RDS, Freitas RL, Calvo F, Muxfeldt M, Leite JP, Landemberger MC, Coimbra NC. Cellular prion protein modulates defensive attention and innate fear-induced behaviour evoked in transgenic mice submitted to an agonistic encounter with the tropical coral snake Oxyrhopus guibei [Internet]. Behavioural Brain Research. 2008 ; 194( 2): 129-137.[citado 2024 maio 29 ] Available from: https://doi.org/10.1016/j.bbr.2008.06.006
Artigo de periodico
Creutzfeldt-Jakob disease associated with a missense mutation at codon 200 of the prion protein gene in Brazil (2007)
Smid, Jerusa; Martins, Vilma Regina; Landemberger, Michele Christine; Riva, Daniele; Anghinah, Renato; Nitrini, Ricardo
Source: Dementia & Neuropsychologia. Unidade: FM
Subjects: GENES, DOENÇA DE PRÍON, DOENÇAS NEURODEGENERATIVAS, TRANSTORNOS COGNITIVOS, PROTEÍNAS (ANÁLISE), BRASIL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SMID, Jerusa et al. Creutzfeldt-Jakob disease associated with a missense mutation at codon 200 of the prion protein gene in Brazil. Dementia & Neuropsychologia, v. 1, n. 2 p. 222-224, 2007Tradução . . Disponível em: https://doi.org/10.1590/s1980-57642008dn10200017. Acesso em: 29 maio 2024.
APA
Smid, J., Martins, V. R., Landemberger, M. C., Riva, D., Anghinah, R., & Nitrini, R. (2007). Creutzfeldt-Jakob disease associated with a missense mutation at codon 200 of the prion protein gene in Brazil. Dementia & Neuropsychologia, 1( 2 p. 222-224). doi:10.1590/s1980-57642008dn10200017
NLM
Smid J, Martins VR, Landemberger MC, Riva D, Anghinah R, Nitrini R. Creutzfeldt-Jakob disease associated with a missense mutation at codon 200 of the prion protein gene in Brazil [Internet]. Dementia & Neuropsychologia. 2007 ; 1( 2 p. 222-224):[citado 2024 maio 29 ] Available from: https://doi.org/10.1590/s1980-57642008dn10200017
Vancouver
Smid J, Martins VR, Landemberger MC, Riva D, Anghinah R, Nitrini R. Creutzfeldt-Jakob disease associated with a missense mutation at codon 200 of the prion protein gene in Brazil [Internet]. Dementia & Neuropsychologia. 2007 ; 1( 2 p. 222-224):[citado 2024 maio 29 ] Available from: https://doi.org/10.1590/s1980-57642008dn10200017
Artigo de periodico
Prion diseases are under compulsory notification in Brazil: surveillance of cases evaluated by biochemical and/or genetic markers from 2005 to 2007 (2007)
Martins, Vilma Regina; Gomes, Hélio Rodrigues; Chimelli, Leila; Rosemberg, Sergio; Landemberger, Michele Christine
Source: Dementia & Neuropsychologia. Unidade: FM
Subjects: MARCADOR MOLECULAR, BIOMARCADORES, DOENÇA DE PRÍON (GENÉTICA), POLIMORFISMO, DOENÇAS NEURODEGENERATIVAS (GENÉTICA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINS, Vilma Regina et al. Prion diseases are under compulsory notification in Brazil: surveillance of cases evaluated by biochemical and/or genetic markers from 2005 to 2007. Dementia & Neuropsychologia, v. 1, n. 4 p. 347-355, 2007Tradução . . Disponível em: https://doi.org/10.1590/s1980-57642008dn10400004. Acesso em: 29 maio 2024.
APA
Martins, V. R., Gomes, H. R., Chimelli, L., Rosemberg, S., & Landemberger, M. C. (2007). Prion diseases are under compulsory notification in Brazil: surveillance of cases evaluated by biochemical and/or genetic markers from 2005 to 2007. Dementia & Neuropsychologia, 1( 4 p. 347-355). doi:10.1590/s1980-57642008dn10400004
NLM
Martins VR, Gomes HR, Chimelli L, Rosemberg S, Landemberger MC. Prion diseases are under compulsory notification in Brazil: surveillance of cases evaluated by biochemical and/or genetic markers from 2005 to 2007 [Internet]. Dementia & Neuropsychologia. 2007 ; 1( 4 p. 347-355):[citado 2024 maio 29 ] Available from: https://doi.org/10.1590/s1980-57642008dn10400004
Vancouver
Martins VR, Gomes HR, Chimelli L, Rosemberg S, Landemberger MC. Prion diseases are under compulsory notification in Brazil: surveillance of cases evaluated by biochemical and/or genetic markers from 2005 to 2007 [Internet]. Dementia & Neuropsychologia. 2007 ; 1( 4 p. 347-355):[citado 2024 maio 29 ] Available from: https://doi.org/10.1590/s1980-57642008dn10400004
Artigo de periodico
Impaired exercise capacity, but unaltered mitochondrial respiration in skeletal or cardiac muscle of mice lacking cellular prion protein (2005)
Nico, Patrícia Barreto Costa; Lobão-Soares, Bruno; Landemberger, Michele Christine; Marques Júnior, Wilson; Tasca, Carla I.; Mello, Carlos Fernando de; Carlotti Júnior, Carlos Gilberto; Walz, Roger; Brentani, Ricardo Renzo; Sakamoto, Américo Ceiki; Bianchin, Marino Muxfeldt
Source: Neuroscience Letters. Unidades: FMRP, FM
Subjects: EXERCÍCIO, RADICAIS LIVRES
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ABNT
NICO, Patrícia Barreto Costa et al. Impaired exercise capacity, but unaltered mitochondrial respiration in skeletal or cardiac muscle of mice lacking cellular prion protein. Neuroscience Letters, v. 388, p. 21-26, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2005.06.033. Acesso em: 29 maio 2024.
APA
Nico, P. B. C., Lobão-Soares, B., Landemberger, M. C., Marques Júnior, W., Tasca, C. I., Mello, C. F. de, et al. (2005). Impaired exercise capacity, but unaltered mitochondrial respiration in skeletal or cardiac muscle of mice lacking cellular prion protein. Neuroscience Letters, 388, 21-26. doi:10.1016/j.neulet.2005.06.033
NLM
Nico PBC, Lobão-Soares B, Landemberger MC, Marques Júnior W, Tasca CI, Mello CF de, Carlotti Júnior CG, Walz R, Brentani RR, Sakamoto AC, Bianchin MM. Impaired exercise capacity, but unaltered mitochondrial respiration in skeletal or cardiac muscle of mice lacking cellular prion protein [Internet]. Neuroscience Letters. 2005 ; 388 21-26.[citado 2024 maio 29 ] Available from: https://doi.org/10.1016/j.neulet.2005.06.033
Vancouver
Nico PBC, Lobão-Soares B, Landemberger MC, Marques Júnior W, Tasca CI, Mello CF de, Carlotti Júnior CG, Walz R, Brentani RR, Sakamoto AC, Bianchin MM. Impaired exercise capacity, but unaltered mitochondrial respiration in skeletal or cardiac muscle of mice lacking cellular prion protein [Internet]. Neuroscience Letters. 2005 ; 388 21-26.[citado 2024 maio 29 ] Available from: https://doi.org/10.1016/j.neulet.2005.06.033

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